Zinc is a trace element, in terms of children, zinc indirectly regulate metabolism through its role, especially for protein synthesis, growth factor production and secretion, such as links to play the impact of its growth.
Zinc deficiency, therefore, the growth of these sectors need to have been clogged. Mucosa and skin is metabolized quickly to update the organization, can occur when zinc deficiency dermatitis, skin lesions, and not easily healed, and the impact of the mucous membrane of the taste for slow performance, the impact of appetite and even anorexia, and abnormal digestion and absorption, which will increase the growth retardation.
Children and adults compared to zinc deficiency relatively easy, fast growth is one of the reasons why children often being choosy food, the habits of a particular kind of food, but for a balanced diet, especially protein intake in terms of a reasonable child, is generally not missing zinc.
Because zinc exists in the protein-containing foods such as meat, dairy, eggs and beans in. Growth retardation has multiple causes, diagnosis means is complex and requires detailed examination of the doctor's diagnosis can be given specific treatment.
If zinc deficiency is not the child, parents are blindly given to the various types of oral zinc products, not only encourage but can not be harmful, too much zinc on liver damage caused by severe cases of hepatitis have jaundice, so zinc should be careful.
Prevention of zinc deficiency disease in children, breast-feeding baby is the best, because human milk is rich in zinc, and easily absorbed from animal foods high zinc content, easy to absorb; therefore appropriate to add rich in infants and young children a variety of trace elements animal liver, egg yolk, meat, fish, mud and other dietary supplements are very necessary in terms of children, adhering to dietary diversification, reasonable arrangements for recipes, to develop good eating habits, not a particular kind of food, not being choosy food, can be prevented by zinc deficiency disease.
Thursday, July 30, 2009
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